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On the other hand, prevention and/or inhibition of Nrf2 pathway by high dose of hormetic Nrf2 modulators would lead to cell death by promoting ROS and doing away with chemoresistance in malignant cells [43]

Treatment with an iron chelator eliminated both protein nitration and liver injury in the absence of LPO [80], suggesting again that under normal conditions, APAP-induced cell death is unlikely to involve ferroptosis [80]