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Molecular and cardiometabolic aspects of ANGPTL3 and apoC-III, as well as inhibition of these targets with monoclonal antibody and nucleic acid therapies, are summarized as background information to descriptions and analyses of recent clinical trials

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Polydatin inhibited TNF--induced apoptosis of skeletal muscle cells through AKT-mediated p38 MAPK and NF-B pathways

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