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An impaired CD4 + T cell response has been suggested to be a hallmark of severe COVID-19 (refs

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Yang Y, Estrada EY, Thompson JF, Liu W, Rosenberg GA (2007) Matrix metalloproteinase-mediated disruption of tight junction proteins in cerebral vessels is reversed by synthetic matrix metalloproteinase inhibitor in focal ischemia in rat

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The activation of SREBP by mTORC1 also increases flux through the pentose phosphate pathway, leading to the production of NADPH and other intermediate metabolites necessary for cellular proliferation and growth

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0.75 M), in the presence or absence of liproxstatin-1 (Lipr

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