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216 RIPK1/3 and MLKL, as central mediators of necroptosis, are promising therapeutic targets in ROS-driven necroptosis
Thus, enhancing TLR3 signaling activation during RT could provide a novel strategy for strengthening abscopal effect to improve the clinical benefits of advanced HCC patients
Antioxidants may be protective against oxidative stress and preserve NO by scavenging ROS and inhibiting oxidized LDL (OxLDL) formation [31]
Mayo Clinic: William Palmer, M.D
