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In a recent study, in addition to elucidating the mechanism of phase transition regulation by a newly identified PTM, namely, the glutathionylation of FUS in both Drosophila and mammalian ALS models, we discovered that GSTO1/GstO2 regulates FUS status
NO system-specific monitoring: Given BPC-157s NO-dependent mechanism, studies designed to assess whether sustained BPC-157 exposure produces effects on blood pressure, vascular reactivity, or markers of NO-mediated oxidative stress would address a mechanistic toxicology question that has not been fully investigated in published BPC-157 research
[DOI] [PMC free article] [PubMed] [Google Scholar] 14.Hahad O., Kuntic M., Kuntic I., Daiber A., Mnzel T
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