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75 Conversely, forced overexpression of CDO1 in breast cancer cells shifted the flux from glutathione synthesis towards cysteine catabolism, consequently increasing ROS levels and leading to reduced cell viability and growth

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In contrast to the limited therapeutic gains observed with fCaCO3-PEG + X-ray irradiation (glucose metabolic regulation alone) or PFC@fSiO2-PEG + X-ray exposure (hypoxia attenuation alone) compared to conventional radiotherapy, PFCE@fCaCO3-PEG markedly enhanced radiotherapy efficacy, resulting in a cure for 4 of 5 mice