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Description
Lifestyle optimization is a promising intervention for enhancing liver regeneration
In summary, combining data from multiple well-defined human cohorts, MASH mouse models and hepatocellular systems, the current study uncovers overproduction of hepatic oxalate due to suppression of AGXT and enhanced LDHA activity that further accelerates MASH through inhibition of PPAR-regulated FAO, enhanced monocyte chemotaxis and ensuing proinflammatory/fibrotic responses
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Rex MR, Williams R, Birsoy K, Ta Llman MS, Stahl M
