activate glutathione Human S Transferase theta 2 (GSTT2) activation kit by CRISPRa 1 Schematic representation of the

Here we show, by studying mice with B cell-specific ablation of the catalytic subunit of glutamate cysteine ligase ( Gclc ), that glutathione synthesis affects homeostasis and differentiation of MZB to a larger extent than FoB, while glutathione-dependent redox control contributes to the metabolic dependencies of FoB

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This pattern reflects the underlying biology rather than treatment failure

Authors acknowledge financial support of the Deanship of academic research at the University of Jordan