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Research indicates: Enhanced glucose transporter expression and membrane translocation Improved insulin sensitivity through AKT pathway modulation Increased metabolic flexibility between glucose and fatty acid utilization Protection against age-related decline in muscle metabolic function Skeletal muscle accounts for approximately 70-85% of insulin-stimulated glucose disposal, making MOTS-c effects in this tissue particularly relevant for systemic metabolic regulation

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Abstract The mammalian target of rapamycin (mTOR), and specifically the mTOR complex 1 (mTORC1) is the central regulator of anabolism in skeletal muscle