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For example, in prostate cancer cells, it elevates the expression of the pro-apoptotic protein Par-4 and activates downstream caspases, while in cervical cancer cells, it reinstates p53 expression, thereby stimulating Bax and other pro-apoptotic members of the Bcl-2 family to induce cell death [109]
Common research themes include: Dual-pathway GH-axis signaling: Studying how GHRH receptor activation and GHSR stimulation influence GH-axis biomarkers and receptor-driven signaling in controlled models
The immune-ferroptosis axis thus represents a double-edged sword in glioblastoma therapy
