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Description
An extracellular matrix-based mechanism of rapid neutrophil extracellular trap formation in response to Candida albicans
This is a reflection of long-term nutrient status not just a snapshot as is seen in serum nutrient tests
5b), whatever the H 2 O 2 or superoxide anions generated at the mitochondrial membrane
These include cumulative oxidative stress, DNA damage, overactivation of the mechanistic target of rapamycin complex 1 (mTORC1) signaling, melanocyte senescence, and reduced production of pigmentation-promoting factors in the hair matrix
