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However, patients with HCC often develop resistance to TKIs and this resistance development is associated with increased IGFBP-1 expression, the aggregation mechanism manifested by TKI-induced hypoxia increasing IGFBP-1 expression through activation of HIF-1 and HIF-2
Quantification of 5-methylcytosine in DNA by the chloroacetaldehyde reaction
A newfound role for sPLA2-IIA in the regulation of gut microbiota has also been discovered
Coates, L
