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This phenomenon can be explained by several mechanisms related to its action in the body: Increased fat metabolism : Carnitine plays a key role in transporting fatty acids to the mitochondria of cells, where they are burned to produce energy

This ATP deficiency disturbs multiple ATP-dependent processes, such as electrolyte transport, substrate utilization, and contractile protein interactions, resulting in impaired myocardial contractility

Thats why we offer our Acetyl-L-Carnitine supplement, the optimal support for cellular and mitochondrial energy

Alpha lipoic acid inhibits neural apoptosis via a mitochondrial pathway in rats following traumatic brain injury