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Treatment Goals: To evaluate treatment success, we must define our goals
Infections may also contribute to HT via molecular mimicry, in which structural or amino acid sequence similarities between microbial and human antigens induce cross-reactive immune responses, accelerating the development of autoimmunity (da Silva et al
Meldonium improves Huntingtons disease mitochondrial dysfunction by restoring peroxisome proliferator-activated receptor gamma coactivator 1alpha expression
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