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As lpd-3 mutants showed probable defective ER-to-PM trafficking of phospholipid, we sought to rescue such organismic phenotypes of lpd-3 mutants by supplementation of phospholipids from various sources and individual constituents of phospholipids, including choline, serine, ethanolamine and fatty acids (unsaturated oleic or saturated stearic acids)

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Summary Given that the present state of knowledge suggests that children with CLP may have a greater risk factor for poor oral health than the general population

Archived from the original on 1 November 2020