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Description
Therefore, targeting Gln metabolism may represent a novel therapeutic strategy against tumor resistance
In contrast, another study reported that exogenous mitochondria can escape endolysosomal compartments and successfully fuse with the endogenous network in human iPSC-derived cardiomyocytes, resulting in sustained increases in ATP production [31]
NAPQI covalently binds to sulfhydryl groups in cellular proteins, especially mitochondrial proteins
Peroxisome proliferator-activated receptor (PPAR): structure, mechanisms of activation and diverse functions
