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Description
aggressive tumors often use FAK signaling to invade surrounding tissue and seed metastases (secondary tumors) By boosting FAKpaxillin activity, BPC-157 might unwittingly give any lurking cancer cells a survival or migration advantage
Just as importantly, we now find that APDs act directly on an additional target in the periphery, pancreatic -cells, where D2R/D3R antagonism by these drugs increases glucagon release
Patch-test for a week
Jiang et al., 2015
