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Description
Resistance mechanisms, such as GPX4 upregulation, compensatory FSP1 activity, and the protective role of specific TME components (e.g., M2 macrophages and Tregs via GPX4 expression), limit the efficacy of ferroptosis
GHK-Cu treatment did not affect histological changes and level of inflammatory cytokines compared to the control group (Figure 5B and 5C)
That is not a subtle decline
doi: 10.1016/S0161-813X(02)00079-7, PMID: [DOI] [PubMed] [Google Scholar] 169.Kim JS, Kim JM, Jeong-Ja O, Jeon BS
