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Recently, ACMSD inhibitors were investigated in the human liver organoid model of metabolic dysfunction-associated steatohepatitis (MASH), an advanced form of liver fibrosis characterized by mitochondrial dysfunction

Phase 2 trial data (Jastreboff et al., NEJM 2023) demonstrated average weight reductions of approximately 1724% over 48 weeks notably higher than currently approved GLP-1 receptor agonists such as semaglutide, though direct comparisons are limited by differences in trial populations and designs

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Subject areas: pathology, public health, human metabolism Graphical abstract Highlights Slightly elevated UCB is a protective host factor against the development of NCDs Humans with elevated UCB had better metabolic health and oxidative stress markers UCB may act as a predictive biomarker of biological aging Pathology