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In a mouse model of OXA-induced liver injury, Robinson et al (10) observed that the expression levels of certain oxidative stress-related genes, including metallothionein 1 (Mt1), heme oxygenase 1 (HO1) and superoxide dismutase 3 (SOD3), were all upregulated
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These insights underscore that addressing immune mechanisms is essential for improving CKD-MBD management, offering a paradigm shift from solely mineral-focused strategies to integrated immune-modulatory approaches
