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Further investigation is warranted to understand the specific mechanisms by which endothelial lipid metabolism maintains phenotypic homeostasis of DR, in consideration of the powerful vaso-regulatory activity of lipid metabolism in other diabetic complications [12]
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COVID-19 is characterized by an excess of pulmonary inflammatory cells such as macrophages, neutrophils, and dendritic cells and elevated levels of inflammatory factors, including IL-2, IL-7, IL-10, G-CSF, IP-10, MCP-1, MIP1A, and TNF-

2009) and clinical studies (Starkey Lewis et al
