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[DOI] [PubMed] [Google Scholar] [52].Yan CY, Greene LA
Kato Y, Kubo Y, Iwata D, et al
In the hypoxic TME, stabilization of HIF-1 not only enhances glycolytic flux through the upregulation of HK2 and PDPK1, but also supports tumor cell motility and invasiveness (77)
In addition to inhibiting NF-B, riboflavin inhibits the release and expression of High-mobility group protein B1, a nuclear factor involved in sepsis regulation and other immune-mediated conditions with a critical role in sepsis-associated multiple organ failure (60)
