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Mechanistic investigations identified NF-B overexpression as critically involved in RA pathogenesis, where its activation triggers cytokine release and upregulates matrix metalloproteinase genes, thereby accelerating extracellular matrix and cartilage degradation
J colloid Interface Sci
Advocates believe peptides can finetune immunity without broadly suppressing it
doi: 10.1017/S0007114510004277 122
