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slc25a39 is necessary for mitochondrial glutathione import in mammalian cells DLAT sustains redox homeostasis and prevent colorectal cancer from ferroptosis by regulating SLC25A39-mediated transport Development of a mouse model

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Nature 547 , 217221 (2017)

slc25a39 is necessary for mitochondrial glutathione import in mammalian cells DLAT sustains redox homeostasis and prevent colorectal cancer from ferroptosis by regulating SLC25A39-mediated transport Development of a mouse model

It is not a steroid, not a hormone, and not a growth hormone releasing peptide

slc25a39 is necessary for mitochondrial glutathione import in mammalian cells DLAT sustains redox homeostasis and prevent colorectal cancer from ferroptosis by regulating SLC25A39-mediated transport Development of a mouse model

1 mg ml 1 in water, pH 7.3) or Lipofectamine 2000 (Life Technologies) and 1,000 ng of plasmid in 100 l of serum-free medium

slc25a39 is necessary for mitochondrial glutathione import in mammalian cells DLAT sustains redox homeostasis and prevent colorectal cancer from ferroptosis by regulating SLC25A39-mediated transport Development of a mouse model

[DOI] [PubMed] [Google Scholar] 34.Yilmaz O., Keser S., Tuzcu M., Guvenc M., Cetintas B., Irtegun S., Tastan H., Sahin K

slc25a39 is necessary for mitochondrial glutathione import in mammalian cells DLAT sustains redox homeostasis and prevent colorectal cancer from ferroptosis by regulating SLC25A39-mediated transport Development of a mouse model

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