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The most promising candidates are pharmacologically innovative next generation TrkB agonists (ANA-12 derivatives) that reproduce BDNF activity and circumvent secretion deficits
Deficiency after LAGB is primarily driven by reduced intake of B12-rich foods and use of acid-suppressing medicines such as PPIs or H2RAs
The gradual supportive nature of micro-dosing aligns with what these conditions require
Interestingly, Veiga-da-Cunha et al
