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Compared to mice with normal Gls1 expression, Gls1-deficient mice exhibited a significant increase in overall survival after radiotherapy, and their levels of natural killer cells and interferon alpha/gamma proteins were significantly higher than those of mice with normal Gls1 expression

Using a Cx3cr1gfp/+ Ccr2rfp/+ transgenic reporter mouse, the temporo-spatial characteristics of monocyte migration into the injured liver and in situ maturation can be followed (see Fig