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doi: 10.1016/S0166-2236(99)01534-9 133 NichollsDG
Pathogenesis 104, 5663
The lack of the main binding interaction with the Asp residue in Ab Pcob may explain the absence of any obvious spectral shift and increase in absorbance at 700 nm upon BV binding that is observed for Sas Pcob (Fig
GCLC desuccinylation protects cancer cells from ferroptosis Our findings that ferroptosis inhibitor Fer-1, instead of other cell death inhibitors (apoptosis, necroptosis), could efficiently protect cells from TBH-induced cell death in SIRT2-knocked down CAKI-1 cells (Fig
