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The next key finding is that IGF-1R, like insulin receptor (IR), is a tyrosine kinase which is activated and autophosphorylated following IGF binding[44]
Humanglutathione S-transferase P1-1 interacts with TRAF2 and regulates TRAF2-ASK1 signals
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[DOI] [PMC free article] [PubMed] [Google Scholar] 56.Pei P., Cheng X., Yu J., Shen J., Li X., Wu J., Wang S., Zhang T
