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Therefore, we summarize the relationships of EGT with several oxidative stress-related diseases, and discuss its possible action mechanisms (Figure 1C)

30 To further confirm that these hepatic abnormalities were directly attributable to NF1 deficiency rather than patient-specific background, we generated an isogenic control by restoring NF1 expression in patient-derived iPSCs using a lentiviral NF1-AON strategy (Supplementary Fig

Bile excretion was studied in six SD rats with BDC