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Endothelial injury generated directly by intracellular viral replication and by ACE2 downregulation, exposing cells to angiotensin II in the absence of the modulator effects of angiotensin 17 (270, 271), and vascular chronic inflammation promoting the development of tissue macrophages overloaded by cholesterol (foam cells), both increase the possibility of acquiring a severe COVID-19 infection (209, 258, 259, 272, 273)

This is thought to be because human digestive enzymes are unable to break the bond between alpha lipoic acid and lysine

That's useful for understanding the molecule, but it doesn't translate into claims about people

[DOI] [PubMed] [Google Scholar] 116.Yldrm Ba F., Bayram D., Arslan B., Armaan I., Yeilot ., iek E., Yorgancgil E