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Consequently, strong CYP3A4 inhibitors (e.g., certain azole antifungals, macrolides) or inducers (e.g., carbamazepine, rifampicin, chronic smoking) may further shift the effective metabolic phenotype and plasma exposure, particularly in patients who are already genetically predisposed to reduced or increased metabolic capacity (Wang et al., 2022)

The Val allele, a product of this SNP, significantly reduces SOD2 activity within the mitochondria via the accelerated degradation of SOD2 Val mRNA

Stem cells, through their paracrine secretions, could target this peripheral pathology by promoting the survival of spiral ganglion neurons or the regeneration of IHC ribbon synapses, thereby restoring balanced input to the brainstem