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The response of the hematopoietic system to high-dose oral folic acid in pernicious anemia patients is very likely the result of entry of UMFA into the hematopoietic progenitor cells via FR-
whereas, HMG CoA synthase-1, an enzyme upstream of HMG CoA reductase, was down-regulated 5070% in WAT of HSL null mice [13]
