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Hu, G., He, M

Similarly, hearts from transgenic mice overexpressing GPx1 had preserved mitochondrial and cardiac function in the presence of doxorubicin, with increased efficiency of mitochondrial respiration and better contractility and diastolic function compared wild type hearts exposed to doxorubicin [44]

There is no solid scientific evidence linking it directly with mood regulation

Once in the nucleus, it binds to specific transcription factors and DNA elements particularly antioxidant response elements (ARE) that are regulated by the stress-responsive factor NRF2. Through this mechanism, MOTS-c can turn on a suite of stress-defense and metabolic genes that help cells adapt to high-fat stress or insulin resistance