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It was found that H 2 S significantly inhibited hypoxia-induced pulmonary VSMCs and reversed chronic hypoxia-induced PH by suppressing endoplasmic reticulum (ER) stress markers in both in vivo and in vitro models ( 2 S was reported to inhibit activating transcription factor 6 (ATF6) and mitigate the hypoxia-induced decline in mitochondrial calcium levels ( 2 S effectively inhibits hypoxia-induced proliferation, migration, and oxidative stress of pulmonary VSMCs

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Around 2,000 were activated, another 2,000 suppressed, many of them linked to inflammation and fibrosis

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