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difficile ) and stimulating the defensive barrier by production of mucin and antimicrobial peptides (defensins and cathelicidins, secreted by specialized cells, Paneth cells and leukocytes in the intestinal crypts) (Guilloteau et al., 2010
Genesis 53 , 474485 (2015)
miR-378 has been proposed to bind ATP6 in mitochondria in the presence of Ago2 and FXR1, leading to a decrease of ATP6 in mouse type 1 diabetic heart (Jagannathan et al., 2015)
Longer-term randomized human trials measuring major cardiovascular outcomes are still needed
