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This phenomenon can be caused by the fact that the released elemental mercury can be re-oxidized in the intracellular compartment by several enzymes, such as catalase [120,121] and re-enter the catalytic GSH-depleting cycle, ultimately leading to a displacement of the intracellular redox potential towards less negative, pro-apoptotic or necrotic values

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A tendon fibroblast study reported that BPC-157 increased growth hormone receptor expression in tendon fibroblasts at both mRNA and protein levels

doi: 10.2174/1871527314666150821104019