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The accumulation of advanced glycation end products (AGEs) can also induce apoptosis (cell death) in fibroblasts, resulting in a significant loss of hyaluronic acid, collagen, and elastin
Frisch, M
Additional hurdles, such as the pathogens slow replication rate, its inhibition of host cell apoptosis, and the sluggish activation and migration of DCs, collectively contribute to the delayed initiation of adaptive immunity
