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Chac2-mediated GSH metabolic reprogramming drives N1-type polarization of neutrophils To determine whether Chac2 is functionally involved in GSH-driven N1-type polarization, we performed both loss- and gain-of-function experiments using HL60-derived neutrophils
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Conclusions and perspectives In recent years, major progress in research on ferroptosis in cancer prevention, diagnostics, prognostics, and treatment has been reported
