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4 Conclusion Therapeutic resistance in multiple myeloma remains a significant obstacle, mainly due to defects in apoptotic signaling
Results showed that VPAC1 and VPAC2 protein levels were significantly higher in PBMC from GD patients compared to healthy donors (Fig
Major CDR L3 contacts are mediated by a set of hydrophobic residues (Trp90, Tyr92-93, and His96) with Tyr93 and His96 contributing the most
doi: 10.1038/sj.bjc.6601623
