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While resistance patterns are changing rapidly and older studies may no longer be valid, there are some low resistance pockets where standard triple therapy may still work

NOS2 expression is suppressed by p53, whereas high NO levels promote p53 stabilization due to its phosphorylation at Ser/Thr residues as well as intracellular p53 accumulation followed by the cell cycle arrest and apoptosis initiation [56, 57]

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In this review, we summarize key findings from secondary analyses of the GOLDEN 3 and 4 studies, and provide a comprehensive overview that may assist both pulmonologists and primary-care providers in their treatment decisions