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Furthermore, the primary sources of free radicals during cerebral ischaemia-reperfusion include calcium-dependent oxidative enzymes such as xanthine oxidase, NADPH oxidase, and nitric oxide synthase, as well as the degradation of membrane and mitochondrial phospholipids [14]

BPC-157 is not legally sold as a dietary supplement or through unverified online channels for human use

SalB promoted Bcl-2 expression and attenuated DOX-induced apoptotic damage in cardiac tissue (109)

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