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Possible mechanisms for the deleterious effects of Hcy on cognitive function are as follows: Hcy leads to DNA breakdown, oxidative damage, and the apoptotic process [7], in addition to as well as excitotoxic cell death by directly activating the neuronal NMDA receptor after formation of homocysteic acid [45]
[DOI] [PMC free article] [PubMed] [Google Scholar] 59.Hou Y.C., Chu C.C., Ko T.L., Yeh C.L., Yeh S.L
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Our growing understanding of redox-mediated cellular events, however, suggest that some ROS is essential for normal cellular processes, such as disulfide bond formation and growth factor receptor-mediated signaling, which requires oxidative inactivation of certain phosphatases
