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The failures in many antioxidant clinical trials likely arise from starting the therapies in the late stages of AD, not monitoring drug levels and markers for the in vivo therapeutic effect of the drug, not utilizing a multi-antioxidant approach that covers both lipophilic and hydrophilic areas of the cell or recycle the oxidized antioxidants back to the reduced state, and not taking into account the basal redox status of the subjects in the trials [10

10.1038/s41467-024-53135-2

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The nephroprotective effect of MS-275 on lipopolysaccharide (LPS)-induced acute kidney injury by inhibiting reactive oxygen species (ROS)-oxidative stress and endoplasmic reticulum stress