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Consistent with this, the lipid peroxidation marker 4-hydroxynonenal (4-HNE) accumulated in both control ( Fbxl5 F/F ) and FBXL5 CD11c mice after APAP treatment (Supplementary Fig

More precisely, scattered curves are generated for each sample in order to evaluate sequencing depth

Abstract The development of ulcerative colitis (UC), a highly prevalent inflammatory bowel disease worldwide, is closely related to the key regulatory role of the PI3K/Akt signaling pathway

The metabolism of APAP has been well-studied and the distributions of its metabolites in the plasma and urine of humans are well-documented [4,6,7], as are the hepatic values in mice and rats [8]