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Its mechanism is competitive inhibition within the NNMT catalytic pocket, blocking the methyl transfer from SAM to nicotinamide
hippocampal GR mRNA restoration (stress-depleted 58% Semax 86% of control)
These findings suggest that although HSP70 is primarily an intracellular chaperone, it can also elicit immune responses when released outside the cell (Mambula and Calderwood, 2006
Riquelme E, Suraokar MB, Rodriguez J, Mino B, Lin HY, Rice DC , Tsao A, Wistuba II
