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Dissociative anesthetics (ketamine and nitrous oxide) impair glutamatergic neurotransmission and mitochondrial function, contributing to excitotoxicity and neurodegeneration

Across mitochondria, NOX enzymes, ER and peroxisomes, physiological H 2 O 2 microgradients encode signalling via reversible cysteine chemistry, whereas spilloverespecially in iron-rich nichestips reactions toward peroxynitrite, Fenton-derived OH, lipid peroxidation and regulated cell death

Some research has suggested that it may enhance tendon and ligament healing, reduce inflammation, and support gastrointestinal health