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In this study, we aim to explore whether chronic ethanol exposure leads to oxidative stress-mediated exacerbation of AD pathology, which in turn results in activation of microglia and neuroinflammatory damage, which ultimately leads to cognitive behavioral disorders

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Melatonin as a potent and inducible endogenous antioxidant: Synthesis and metabolism

Figure 5 4 Promising therapeutical reagents for SLE by inhibiting ferroptosis Given that several pathophysiological features of ferroptosis, including iron accumulation, mitochondrial dysfunction, overproduction of lipid peroxidation and suppression xCT-GPX4 pathway are potentially implicated in SLE progression, reversing those features may be beneficial to SLE disease