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T., Cozzolino, M
Weber, 2012)
S6D), and decreased BrdU + proliferation cells in HG (Fig
Akt is deactivated and Bcl-2-associated death promoter (BAD) phosphorylation is lost in one pathway due to a stalled survival pathway, whereas Fas cell surface death receptor (FAS) is upregulated through stimulation of c-Jun-N-terminal kinase, which activates caspase-8, phosphorylates Fas-associated protein with death domain (FADD), and truncates BH3-interacting domain death agonist (BID)
