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EGFR deletion also decreased the abundance of F4/80 hi macrophages, particularly the Spp1 + and C1qc + subsets, and reduced inflammatory pathways like TGF, IFN, and JAK/STAT signaling in myeloid cells
(2016) reported that the absence of BMAL1 in mice not only disrupts circadian rhythms but also increases oxidative stress, impairs mitochondrial function, and perturbs metabolic pathways, pointing to a clock-independent role for BMAL1 in maintaining redox balance, proteostasis, and tissue integrity
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