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Mechanistically, CEBPB orchestrates GC malignancy through intricate interactions with metabolic, inflammatory, and transcriptional networks
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demonstrated that 2060% of 9(10)-EpODA was converted to 9,10-DiHODA, and 50% of 12-HpODA was degraded, while 12-HODA and 12-oxo-ODA were unaltered

Large-scale clinical trials have demonstrated that elevated plasma LDL-C levels increase the risk of ASCVD [4850], and this biological impact is closely associated with the level and duration of exposure [51, 52]